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1. Epithelial-derived interleukin-23 promotes oral mucosal immunopathology

3. Response to Comments on “Aberrant type 1 immunity drives susceptibility to mucosal fungal infections”

4. Aberrant type 1 immunity drives susceptibility to mucosal fungal infections

11. On-going Mechanical Damage from Mastication Drives Homeostatic Th17 Cell Responses at the Oral Barrier

13. Fibrin is a critical regulator of neutrophil effector function at the oral mucosal barrier

18. Fibrin is a critical regulator of neutrophil effector function at mucosal barrier sites

22. Secretory leukocyte protease inhibitor mediates non-redundant functions necessary for normal wound healing

26. A dysbiotic microbiome triggers T H 17 cells to mediate oral mucosal immunopathology in mice and humans

27. Human defects in STAT3 promote oral mucosal fungal and bacterial dysbiosis

28. On-going mechanical damage from mastication drives homeostatic Th17 responses at the oral barrier

29. Microbiota-independent mechanisms shape Th17 homeostatic responses at the oral barrier.

30. A dysbiotic microbiome triggers TH17 cells to mediate oral mucosal immunopathology in mice and humans.

31. Neutrophil extracellular traps and extracellular histones potentiate IL-17 inflammation in periodontitis

32. Subgingival Microbial Communities in Leukocyte Adhesion Deficiency and Their Relationship with Local Immunopathology

33. Defective Neutrophil Recruitment in Leukocyte Adhesion Deficiency Type I Disease Causes Local IL-17–Driven Inflammatory Bone Loss

35. Tumor necrosis factor‐alpha (TNF‐α) is a therapeutic target for impaired cutaneous wound healing

38. CORRECTION

43. Human Immunodeficiency Virus Type 1-Induced Macrophage Gene Expression Includes the p21 Gene, a Target for Viral Regulation

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