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1. An arrayed CRISPR screen of primary B cells reveals the essential elements of the antibody secretion pathway

2. Epigenetic modulators of B cell fate identified through coupled phenotype-transcriptome analysis

3. Mining the Plasma Cell Transcriptome for Novel Cell Surface Proteins

4. A point mutation in the Ncr1 signal peptide impairs the development of innate lymphoid cell subsets

5. Environmental sensing by mature B cells is controlled by the transcription factors PU.1 and SpiB

6. Investigating the Antigen Specificity of Multiple Sclerosis Central Nervous System-Derived Immunoglobulins

7. The transcription factors IRF8 and PU.1 negatively regulate plasma cell differentiation

8. Autoantibodies produced at the site of tissue damage provide evidence of humoral autoimmunity in inclusion body myositis.

9. Structural Basis for Apoptosis Inhibition by Epstein-Barr Virus BHRF1

10. A novel BH3 ligand that selectively targets Mcl-1 reveals that apoptosis can proceed without Mcl-1 degradation

11. Epstein-Barr virus infection is not a characteristic feature of multiple sclerosis brain.

12. Functionally distinct roles for different miR-155 expression levels through contrasting effects on gene expression, in acute myeloid leukaemia

13. The histone acetyltransferase KAT6B is required for hematopoietic stem cell development and function.

14. An arrayed CRISPR screen of primary B cells reveals the essential elements of the antibody secretion pathway.

15. Epigenetic modulators of B cell fate identified through coupled phenotype-transcriptome analysis.

16. The transcription factor IRF4 represses proapoptotic BMF and BIM to licence multiple myeloma survival.

17. OBF1 and Oct factors control the germinal center transcriptional program.

18. IRF4 Activity Is Required in Established Plasma Cells to Regulate Gene Transcription and Mitochondrial Homeostasis.

19. New players in the gene regulatory network controlling late B cell differentiation.

20. IMiDs prime myeloma cells for daratumumab-mediated cytotoxicity through loss of Ikaros and Aiolos.

21. A point mutation in the Ncr1 signal peptide impairs the development of innate lymphoid cell subsets.

22. Mining the Plasma Cell Transcriptome for Novel Cell Surface Proteins.

23. Environmental sensing by mature B cells is controlled by the transcription factors PU.1 and SpiB.

24. Functionally distinct roles for different miR-155 expression levels through contrasting effects on gene expression, in acute myeloid leukaemia.

25. NFκB1 is essential to prevent the development of multiorgan autoimmunity by limiting IL-6 production in follicular B cells.

26. Investigating the Antigen Specificity of Multiple Sclerosis Central Nervous System-Derived Immunoglobulins.

27. Transcriptional profiling of mouse B cell terminal differentiation defines a signature for antibody-secreting plasma cells.

28. The transcription factors IRF8 and PU.1 negatively regulate plasma cell differentiation.

29. Transcription factor IRF4 regulates germinal center cell formation through a B cell-intrinsic mechanism.

30. Protein array-based profiling of CSF identifies RBPJ as an autoantigen in multiple sclerosis.

31. Autoantibodies produced at the site of tissue damage provide evidence of humoral autoimmunity in inclusion body myositis.

32. Related B cell clones that populate the CSF and CNS of patients with multiple sclerosis produce CSF immunoglobulin.

33. Related B cell clones populate the meninges and parenchyma of patients with multiple sclerosis.

34. Structural basis for apoptosis inhibition by Epstein-Barr virus BHRF1.

35. A unique antibody gene signature is prevalent in the central nervous system of patients with multiple sclerosis.

36. The microenvironment of germ cell tumors harbors a prominent antigen-driven humoral response.

37. A novel BH3 ligand that selectively targets Mcl-1 reveals that apoptosis can proceed without Mcl-1 degradation.

39. Interleukin 15-mediated survival of natural killer cells is determined by interactions among Bim, Noxa and Mcl-1.

40. Apoptosis initiated when BH3 ligands engage multiple Bcl-2 homologs, not Bax or Bak.

41. The BH3 mimetic ABT-737 targets selective Bcl-2 proteins and efficiently induces apoptosis via Bak/Bax if Mcl-1 is neutralized.

42. CD45 links the B cell receptor with cell survival and is required for the persistence of germinal centers.

43. Life in the balance: how BH3-only proteins induce apoptosis.

44. Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins.

45. Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function.

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