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1. Genomic and Molecular Analyses Identify Molecular Subtypes of Pancreatic Cancer Recurrence

2. Targeting DNA Damage Response and Replication Stress in Pancreatic Cancer

3. HNF4A and GATA6 Loss Reveals Therapeutically Actionable Subtypes in Pancreatic Cancer

4. Identification of unique neoantigen qualities in long-term survivors of pancreatic cancer

5. Genomic and Molecular Analyses Identify Molecular Subtypes of Pancreatic Cancer Recurrence

6. Whole genomes redefine the mutational landscape of pancreatic cancer

7. DNA methylation patterns identify subgroups of pancreatic neuroendocrine tumors with clinical association

8. Pancreatic cancer genomes reveal aberrations in axon guidance pathway genes

9. The deubiquitinase USP9X suppresses pancreatic ductal adenocarcinoma

10. Targeting DNA Damage Response and Replication Stress in Pancreatic Cancer

11. HNF4A and GATA6 Loss Reveals Therapeutically Actionable Subtypes in Pancreatic Cancer

13. Distribution and pathological features of pancreatic, ampullary, biliary and duodenal cancers resected with pancreaticoduodenectomy

16. HNF4Aand GATA6Loss Reveals Therapeutically Actionable Subtypes in Pancreatic Cancer

18. BOOK REVIEWS.

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