41 results on '"Yoo, Jae Hyuk"'
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2. Correction: Activation of NFAT by HGF and IGF-1 via ARF6 and its effector ASAP1 promotes uveal melanoma metastasis
3. Small GTPase ARF6 controls VEGFR2 trafficking and signaling in diabetic retinopathy
4. Data from The Small GTPase ARF6 Activates PI3K in Melanoma to Induce a Prometastatic State
5. Figure S2 from The Small GTPase ARF6 Activates PI3K in Melanoma to Induce a Prometastatic State
6. Figure S7 from Chloroquine Sensitizes GNAQ/11-mutated Melanoma to MEK1/2 Inhibition
7. Supplementary Methods from Chloroquine Sensitizes GNAQ/11-mutated Melanoma to MEK1/2 Inhibition
8. Table S1 from The Small GTPase ARF6 Activates PI3K in Melanoma to Induce a Prometastatic State
9. Type I Interferon Signaling Is Critical During the Innate Immune Response to HSV-1 Retinal Infection
10. Activation of NFAT by HGF and IGF-1 via ARF6 and its effector ASAP1 promotes uveal melanoma metastasis
11. PI3Ka-Akt1-mediated Prdm4 induction in adipose tissue increases energy expenditure, inhibits weight gain, and improves insulin resistance in diet-induced obese mice
12. Chloroquine SensitizesGNAQ/11-mutated Melanoma to MEK1/2 Inhibition
13. Pathogen- and NaCl-induced expression of the SCaM-4 promoter is mediated in part by a GT-1 box that interacts with a GT-1-like transcription factor (1)
14. Abstract 6078: Suppression of tumor-intrinsic IFNγ signaling in ARF6-mediated metastatic progression of melanoma
15. Abstract 1890: Chloroquine synergizes with MEK1/2 targeted therapy through dual YAP and lysosomal inhibition in GNAQ/11 mutant uveal melanoma
16. Mouse models of uveal melanoma: Strengths, weaknesses, and future directions
17. Isolation of a Calmodulin-binding Transcription Factor from Rice (Oryza sativa L.)
18. Characterization of a stamen-specific cDNA encoding a novel plant defensin in Chinese cabbage
19. Tris DBA palladium is an orally available inhibitor of GNAQ mutant uveal melanoma in vivo
20. The Small GTPase ARF6 Activates PI3K in Melanoma to Induce a Prometastatic State
21. Sulfuretin Prevents Obesity and Metabolic Diseases in Diet Induced Obese Mice
22. Abstract 5157: ARF6 controls WNT5A receptor internalization to promote uveal melanoma invasion and metastasis
23. Abstract 4367: The small GTPase ARF6 is necessary for melanomagenesis
24. ARF6 is a Targetable Node that Orchestrates Signalling of Multiple Oncogenic Pathways in Cutaneous and Uveal Melanoma
25. The small GTPase ARF6 regulates protein trafficking to control cellular function during development and in disease
26. The small GTPase ARF6 regulates protein trafficking to control cellular function during development and in disease.
27. ARF6 Is an Actionable Node that Orchestrates Oncogenic GNAQ Signaling in Uveal Melanoma
28. An S-locus receptor-like kinase in plasma membrane interacts with calmodulin in Arabidopsis
29. The Small GTPase ARF6 Stimulates β-Catenin Transcriptional Activity During WNT5A-Mediated Melanoma Invasion and Metastasis
30. An S-locus receptor-like kinase in plasma membrane interacts with calmodulin inArabidopsis
31. Regulation of MAPK Phosphatase 1 (AtMKP1) by Calmodulin in Arabidopsis
32. Identification of a Calmodulin-binding NAC Protein as a Transcriptional Repressor in Arabidopsis
33. Interaction of a Dual‐Specificity Protein Phosphatase with Calmodulin in Regulating its Activity
34. Arabidopsis ubiquitin-specific protease 6 (AtUBP6) interacts with calmodulin
35. WRKY group IId transcription factors interact with calmodulin
36. Direct Interaction of a Divergent CaM Isoform and the Transcription Factor, MYB2, Enhances Salt Tolerance in Arabidopsis
37. Regulation of the Dual Specificity Protein Phosphatase, DsPTP1, through Interactions with Calmodulin
38. Identification of Calmodulin Isoform-specific Binding Peptides from a Phage-displayed Random 22-mer Peptide Library
39. Mlo, a Modulator of Plant Defense and Cell Death, Is a Novel Calmodulin-binding Protein
40. Chloroquine Sensitizes GNAQ/11 -mutated Melanoma to MEK1/2 Inhibition.
41. Pathogenesis-related gene expression by specific calmodulin isoforms is dependent on NIM1, a key regulator of systemic acquired resistance.
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