32 results on '"Yu, Jungeun"'
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2. NOTCH2 sensitizes the chondrocyte to the inflammatory response of tumor necrosis factor α
3. Notch and the regulation of osteoclast differentiation and function
4. Cbl-PI3K interaction regulates Cathepsin K secretion in osteoclasts
5. Induction of the Hajdu-Cheney Syndrome Mutation in CD19 B Cells in Mice Alters B-Cell Allocation but Not Skeletal Homeostasis
6. Loss of Cbl-PI3K interaction modulates the periosteal response to fracture by enhancing osteogenic commitment and differentiation
7. Use of antisense oligonucleotides to target Notch3 in skeletal cells
8. 1,3-Dibenzyl-5-Fluorouracil Prevents Ovariectomy-Induced Bone Loss by Suppressing Osteoclast Differentiation
9. Hairy and enhancer of split 1 is a primary effector of NOTCH2 signaling and induces osteoclast differentiation and function
10. D-chiro-inositol Negatively Regulates the Formation of Multinucleated Osteoclasts by Down-Regulating NFATc1
11. T-Cell Death Associated Gene 51 Is a Novel Negative Regulator of PPARγ That Inhibits PPARγRXRα Heterodimer Formation in Adipogenesis
12. Activation of Notch3 in osteoblasts/osteocytes causes compartment-specific changes in bone remodeling
13. Nuclear factor of activated T cells 1 and 2 are required for vertebral homeostasis
14. Antisense oligonucleotides targeting Notch2 ameliorate the osteopenic phenotype in a mouse model of Hajdu-Cheney syndrome
15. A PDGFRβ-PI3K signaling axis mediates periosteal cell activation during fracture healing
16. The Hajdu Cheney mutation sensitizes mice to the osteolytic actions of tumor necrosis factor α
17. TDAG51 is a crucial regulator of maternal care and depressive-like behavior after parturition
18. An antibody to Notch3 reverses the skeletal phenotype of lateral meningocele syndrome in male mice
19. PDGF Modulates BMP2-Induced Osteogenesis in Periosteal Progenitor Cells
20. An antibody to Notch3 reverses the skeletal phenotype of lateral meningocele syndrome in male mice.
21. The lateral meningocele syndrome mutation causes marked osteopenia in mice
22. Nuclear factor of activated T cells 2 is required for osteoclast differentiation and function in vitro but not in vivo
23. Glucocorticoids inhibit notch target gene expression in osteoblasts
24. The Hajdu Cheney Mutation Is a Determinant of B-Cell Allocation of the Splenic Marginal Zone
25. Sustained Notch2 signaling in osteoblasts, but not in osteoclasts, is linked to osteopenia in a mouse model of Hajdu-Cheney syndrome
26. An Antibody to Notch2 Reverses the Osteopenic Phenotype of Hajdu-Cheney Mutant Male Mice
27. Interaction of Tumor Necrosis Factor Receptor-associated Factor 6 (TRAF6) and Vav3 in the Receptor Activator of Nuclear Factor κB (RANK) Signaling Complex Enhances Osteoclastogenesis
28. Increased periosteal expansion, Osterix expression and osteogenic potential upon bone injury during perturbed PI3K signaling
29. Tumor Necrosis Factor (TNF) Receptor-associated Factor (TRAF)-interacting Protein (TRIP) Negatively Regulates the TRAF2 Ubiquitin-dependent Pathway by Suppressing the TRAF2-Sphingosine 1-Phosphate (S1P) Interaction
30. Secretion of a Truncated Osteopetrosis-associated Transmembrane Protein 1 (OSTM1) Mutant Inhibits Osteoclastogenesis through Down-regulation of the B Lymphocyte-induced Maturation Protein 1 (BLIMP1)-Nuclear Factor of Activated T Cells c1 (NFATc1) Axis
31. Protein arginine methyltransferase 1 regulates herpes simplex virus replication through ICP27 RGG-box methylation
32. T-Cell Death Associated Gene 51 Is a Novel Negative Regulator of PPARγ That Inhibits PPARγ-RXRα Heterodimer Formation in Adipogenesis
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