39 results on '"Yu, Xilan"'
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2. Phosphoglycerate dehydrogenase activates PKM2 to phosphorylate histone H3T11 and attenuate cellular senescence
3. Phosphorylation of Jhd2 by the Ras-cAMP-PKA(Tpk2) pathway regulates histone modifications and autophagy
4. SESAME-catalyzed H3T11 phosphorylation inhibits Dot1-catalyzed H3K79me3 to regulate autophagy and telomere silencing
5. Acetylation-dependent SAGA complex dimerization promotes nucleosome acetylation and gene transcription
6. Specific regulation of epigenome landscape by metabolic enzymes and metabolites.
7. The SESAME complex regulates cell senescence through the generation of acetyl-CoA
8. Metabolic regulation of telomere silencing by SESAME complex-catalyzed H3T11 phosphorylation
9. Cla4 phosphorylates histone methyltransferase Set1 to prevent its degradation by the APC/C Cdh1 complex
10. Transcriptional regulation of autophagy by chromatin remodeling complex and histone variant
11. The TORC1 activates Rpd3L complex to deacetylate Ino80 and H2A.Z and repress autophagy
12. Regulation of telomere silencing by the core histones–autophagy–Sir2 axis
13. Hepatitis C virus and its protein NS4B activate the cancer-related STAT3 pathway via the endoplasmic reticulum overload response
14. Transcriptional responses of Pseudomonas syringae to growth in epiphytic versus apoplastic leaf sites
15. Interplay Between Glucose Metabolism and Chromatin Modifications in Cancer
16. Histone acetyltransferase Gcn5 regulates gene expression by promoting the transcription of histone methyltransferase SET1
17. Glycolysis regulates gene expression by promoting the crosstalk between H3K4 trimethylation and H3K14 acetylation in Saccharomyces cerevisiae
18. Exogenous pyruvate represses histone gene expression and inhibits cancer cell proliferation via the NAMPT–NAD+–SIRT1 pathway
19. Set1-catalyzed H3K4 trimethylation antagonizes the HIR/Asf1/Rtt106 repressor complex to promote histone gene expression and chronological life span
20. Reciprocal Regulation of Metabolic Reprogramming and Epigenetic Modifications in Cancer
21. Exogenous pyruvate represses histone gene expression and inhibits cancer cell proliferation via the NAMPT–NAD+–SIRT1 pathway.
22. Japanese encephalitis virus induces apoptosis by inhibiting Foxo signaling pathway
23. Regulation of DNA replication-coupled histone gene expression
24. Inhibition Activity of Herpes Virus (HSV) Replication by α-TIF siRNA-Loaded PLGA-TPGS Nanoparticles In Vitro and In Vivo
25. Regulation of SESAME-mediated H3T11 phosphorylation by glycolytic enzymes and metabolites
26. Anti-tumor activity of metformin: from metabolic and epigenetic perspectives
27. Study of Blending Mechanism of Bio-Binder and Petroleum Asphalt
28. Transcriptional Analysis of the Global Regulatory Networks Active in Pseudomonas syringae during Leaf Colonization
29. Host–virus interactions: from the perspectives of epigenetics
30. Physiological and Transcriptional Responses to Osmotic Stress of Two Pseudomonas syringae Strains That Differ in Epiphytic Fitness and Osmotolerance
31. The expanding roles of endoplasmic reticulum stress in virus replication and pathogenesis
32. Glycine Betaine Catabolism Contributes to Pseudomonas syringae Tolerance to Hyperosmotic Stress by Relieving Betaine-Mediated Suppression of Compatible Solute Synthesis
33. Interaction networks of hepatitis C virus NS4B: implications for antiviral therapy
34. Characterization of multiple regulatory networks and responses to environmental signals in Pseudomonas syringae during its association with plants
35. Hepatitis C virus NS4B induces unfolded protein response and endoplasmic reticulum overload response-dependent NF-κB activation
36. The expanding roles of endoplasmic reticulum stress in virus replication and pathogenesis.
37. Transcriptional Analysis of the Global Regulatory Networks Active in Pseudomonas syringaeduring Leaf Colonization
38. Anti-tumor activity of metformin: from metabolic and epigenetic perspectives.
39. Hepatitis C virus NS4B induces unfolded protein response and endoplasmic reticulum overload response-dependent NF-kappaB activation.
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