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4. Cardiac fibrosis as a predictor for sudden cardiac death after transcatheter aortic valve implantation

6. Single-nucleus transcriptomics reveals adrenergic and STAT3 signalling in paradoxical low-flow low-gradient-specific cardiomyocyte subclusters: implications for aortic stenosis pathogenesis and treatment.

7. Postnatal expression of the lysine methyltransferase SETD1B is essential for learning and the regulation of neuron‐enriched genes

11. Low-flow in aortic valve stenosis patients with reduced ejection fraction does not depend on left ventricular function

13. An Alternative Mechanism of Subcellular Iron Uptake Deficiency in Cardiomyocytes

15. An Alternative Mechanism of Subcellular Iron Uptake Deficiency in Cardiomyocytes

16. Pharmacological induction of hypoxia-inducible transcription factor ARNT attenuates chronic kidney failure

17. Aortic valve calcification and myocardial fibrosis determine outcome following transcatheter aortic valve replacement

18. Additional file 1 of miR-132-3p and KLF7 as novel regulators of aortic stiffening-associated EndMT in type 2 diabetes mellitus

22. Postnatal expression of the lysine methyltransferase SETD1B is essential for learning and the regulation of neuron‐enriched genes

24. Postnatal SETD1B is essential for learning and the regulation of neuronal plasticity genes

25. Methylation determines fibroblast activation and fibrogenesis in the kidney

26. Endothelial-to-mesenchymal transition contributes to cardiac fibrosis

28. SETD1B controls cognitive function via cell type specific regulation of neuronal identity genes

29. Endothelial-to-mesenchymal transition compromises vascular integrity to induce Myc-mediated metabolic reprogramming in kidney fibrosis

31. Interdisciplinary Research on Aortic Valve Stenosis: A Longitudinal Collection of Biospecimens and Clinical Data of Patients Undergoing Transcatheter Aortic Valve Replacement

35. Sarcoplasmic reticulum calcium leak contributes to arrhythmia but not to heart failure progression

38. Association of circulating angiogenesis inhibitors and asymmetric dimethyl arginine with coronary plaque burden

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