13 results on '"Zhao, Zai Hao"'
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2. Reduced nNOS activity is responsible for impaired fatty acid–dependent mitochondrial oxygen consumption in atrial myocardium from hypertensive rat
3. Enlargement of opisthenar microcirculatory area predicts impaired heart function in severe acute coronary syndrome patients
4. S-nitrosylation of transglutaminase 2 impairs fatty acid-stimulated contraction in hypertensive cardiomyocytes
5. Neuronal nitric oxide synthase modulation of intracellular Ca2+ handling overrides fatty acid potentiation of cardiac inotropy in hypertensive rats
6. Negligible effect of eNOS palmitoylation on fatty acid regulation of contraction in ventricular myocytes from healthy and hypertensive rats
7. Gender Differences in the Correlations Between Immune Cells and Organ Damage Indexes of Acute Myocardial Infarction Patients
8. Correction to: Reduced nNOS activity is responsible for impaired fatty acid–dependent mitochondrial oxygen consumption in atrial myocardium from hypertensive rat
9. Cardiac inotropy, lusitropy, and Ca2+ handling with major metabolic substrates in rat heart
10. Low K+ current in arterial myocytes with impaired K+-vasodilation and its recovery by exercise in hypertensive rats
11. Assessment of Myofilament Ca2+ Sensitivity Underlying Cardiac Excitation-contraction Coupling
12. Modulation of L-type Ca2+ channel activity by neuronal nitric oxide synthase and myofilament Ca2+ sensitivity in cardiac myocytes from hypertensive rat
13. Distinct roles of neuronal nitric oxide synthase in metabolic substrates‐induced abnormal Ca 2+ handling and cardiac arrhythmias in normal and hypertensive rats
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