14 results on '"de la Barrera, Silvia S."'
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2. Role Played by the Programmed Death-1—Programmed Death Ligand Pathway during Innate Immunity against Mycobacterium tuberculosis
3. In Tuberculous Pleural Effusions, Activated Neutrophils Undergo Apoptosis and Acquire a Dendritic Cell-Like Phenotype
4. Mycobacterium tuberculosis-Induced Activation Accelerates Apoptosis in Peripheral Blood Neutrophils from Patients with Active Tuberculosis
5. Clinical Isolates ofMycobacterium tuberculosisDiffer in Their Ability to Induce Respiratory Burst and Apoptosis in Neutrophils as a Possible Mechanism of Immune Escape
6. Role Played by the Programmed Death‐1–Programmed Death Ligand Pathway during Innate Immunity againstMycobacterium tuberculosis
7. NK cells from tuberculous pleurisy express high ICAM‐1 levels and exert stimulatory effect on local T cells
8. Mycobacterium tuberculosis -Induced Gamma Interferon Production by Natural Killer Cells Requires Cross Talk with Antigen-Presenting Cells Involving Toll-Like Receptors 2 and 4 and the Mannose Receptor in Tuberculous Pleurisy
9. Increased Susceptibility to Apoptosis of CD56dimCD16+ NK Cells Induces the Enrichment of IFN-γ-Producing CD56bright Cells in Tuberculous Pleurisy
10. Mycobacterium tuberculosisTriggers Apoptosis in Peripheral Neutrophils Involving Toll-Like Receptor 2 and p38 Mitogen Protein Kinase in Tuberculosis Patients
11. Clinical Isolates of Mycobacterium tuberculosis Differ in Their Ability to Induce Respiratory Burst and Apoptosis in Neutrophils as a Possible Mechanism of Immune Escape.
12. HIV infection and natural killer cytotoxicity in hemophilic patients
13. Increased susceptibility to apoptosis of CD56dimCD16+ NK cells induces the enrichment of IFN-gamma-producing CD56bright cells in tuberculous pleurisy.
14. Mycobacterium tuberculosis triggers apoptosis in peripheral neutrophils involving toll-like receptor 2 and p38 mitogen protein kinase in tuberculosis patients.
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