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3. Biallelic NFATC1 mutations cause an inborn error of immunity with impaired CD8+ T-cell function and perturbed glycolysis

4. Redox regulation of metabolism in asthma: new insights into the roles of Glutathione-S-transferase P

6. Glutathione-S-transferase P promotes glycolysis in asthma in association with oxidation of pyruvate kinase M2

7. Glutathione S-transferases and their implications in the lung diseases asthma and chronic obstructive pulmonary disease: Early life susceptibility?

8. Redox regulation of metabolism in asthma

9. Dysregulation of Pyruvate Kinase M2 Promotes Inflammation in a Mouse Model of Obese Allergic Asthma

10. Downregulation of epithelial DUOX1 in chronic obstructive pulmonary disease

12. Pyruvate Kinase M2 Promotes Expression of Proinflammatory Mediators in House Dust Mite–Induced Allergic Airways Disease

13. Dysregulation of the glutaredoxin/S-glutathionylation redox axis in lung diseases

14. Peroxiredoxins and Beyond; Redox Systems Regulating Lung Physiology and Disease

15. Glutathione-S-transferase π promotes interleukin-1β induced pulmonary inflammation in association with S-glutathionylation of the glycolysis regulator, pyruvate kinase M2

16. S-glutathionylation of pyruvate kinase M2 is associated with metabolic reprogramming and cytokine production in the development of allergic obese airway disease

17. IL-1/inhibitory κB kinase ε–induced glycolysis augment epithelial effector function and promote allergic airways disease

21. Biallelic NFATC1mutations cause an inborn error of immunity with impaired CD8+ T-cell function and perturbed glycolysis

22. Glutathionylation chemistry promotes interleukin-1 beta-mediated glycolytic reprogramming and pro-inflammatory signaling in lung epithelial cells.

23. LTβR deficiency causes lymph node aplasia and impaired B cell differentiation.

24. Dysregulation of the glutaredoxin/ S- glutathionylation redox axis in lung diseases.

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