22 results on '"van den Berk, Paul C. M."'
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2. A C57BL/6J Fancg-KO Mouse Model Generated by CRISPR/Cas9 Partially Captures the Human Phenotype
3. Mammalian life depends on two distinct pathways of DNA damage tolerance
4. Huwe1 supports B-cell development, B-cell-dependent immunity, somatic hypermutation and class switch recombination by regulating proliferation
5. Division of labor within the DNA damage tolerance system reveals non-epistatic and clinically actionable targets for precision cancer medicine
6. Somatic Hypermutation of Immunoglobulin Genes: Lessons from Proliferating Cell Nuclear ${\text{Antigen}}^{K164R} $ Mutant Mice
7. The Widely Used Antihelmintic Drug Albendazole is a Potent Inducer of Loss of Heterozygosity
8. Lysis of Syngeneic Tumor B Cells by Autoreactive Cytotoxic T Lymphocytes Specific for a CD 19 Antigen-Derived Synthetic Peptide
9. PrimPol prevents APOBEC/AID family mediated DNA mutagenesis.
10. Lysine Residue 185 of Rad1 Is a Topological but Not a Functional Counterpart of Lysine Residue 164 of PCNA
11. The Fanconi Anemia Core Complex Is Dispensable during Somatic Hypermutation and Class Switch Recombination
12. Dual role of proliferating cell nuclear antigen monoubiquitination in facilitating Fanconi anemia-mediated interstrand crosslink repair.
13. Precision cancer therapy: profiting from tumor specific defects in the DNA damage tolerance system.
14. Quantitative analysis by next generation sequencing of hematopoietic stem and progenitor cells (LSK) and of splenic B cells transcriptomes from wild-type and Usp3-knockout mice.
15. Roles of PCNA ubiquitination and TLS polymerases κ and η in the bypass of methyl methanesulfonate-induced DNA damage.
16. Tight regulation of ubiquitin-mediated DNA damage response by USP3 preserves the functional integrity of hematopoietic stem cells.
17. Differential programming of B cells in AID deficient mice.
18. PCNA ubiquitination-independent activation of polymerase η during somatic hypermutation and DNA damage tolerance.
19. HLTF and SHPRH are not essential for PCNA polyubiquitination, survival and somatic hypermutation: existence of an alternative E3 ligase.
20. Dependence of nucleotide substitutions on Ung2, Msh2, and PCNA-Ub during somatic hypermutation.
21. A/T mutagenesis in hypermutated immunoglobulin genes strongly depends on PCNAK164 modification.
22. Immunogenicity, including vitiligo, and feasibility of vaccination with autologous GM-CSF-transduced tumor cells in metastatic melanoma patients.
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