Indoor air sampling for fine particulate matter and black carbon in industrial communities in Pittsburgh.

Impacts of industrial emissions on outdoor air pollution in nearby communities are well-documented. Fewer studies, however, have explored impacts on indoor air quality in these communities. Because persons in northern climates spend a majority of their time indoors, understanding indoor exposures, and the role of outdoor air pollution in shaping such exposures, is a priority issue. Braddock and Clairton, Pennsylvania, industrial communities near Pittsburgh, are home to an active steel mill and coke works, respectively, and the population experiences elevated rates of childhood asthma. Twenty-one homes were selected for 1-week indoor sampling for fine particulate matter (PM 2.5 ) and black carbon (BC) during summer 2011 and winter 2012. Multivariate linear regression models were used to examine contributions from both outdoor concentrations and indoor sources. In the models, an outdoor infiltration component explained 10 to 39% of variability in indoor air pollution for PM 2.5 , and 33 to 42% for BC. For both PM 2.5 models and the summer BC model, smoking was a stronger predictor than outdoor pollution, as greater pollutant concentration increases were identified. For winter BC, the model was explained by outdoor pollution and an open windows modifier. In both seasons, indoor concentrations for both PM 2.5 and BC were consistently higher than residence-specific outdoor concentration estimates. Mean indoor PM 2.5 was higher, on average, during summer (25.8 ± 22.7 μg/m 3 ) than winter (18.9 ± 13.2 μg/m 3 ). Contrary to the study's hypothesis, outdoor concentrations accounted for only little to moderate variability (10 to 42%) in indoor concentrations; a much greater proportion of PM 2.5 was explained by cigarette smoking. Outdoor infiltration was a stronger predictor for BC compared to PM 2.5 , especially in winter. Our results suggest that, even in industrial communities of high outdoor pollution concentrations, indoor activities – particularly cigarette smoking – may play a larger role in shaping indoor exposures. [ABSTRACT FROM AUTHOR]