Clk1 deficiency promotes neuroinflammation and subsequent dopaminergic cell death through regulation of microglial metabolic reprogramming.

MLA

Gu, Ruinan, et al. “Clk1 Deficiency Promotes Neuroinflammation and Subsequent Dopaminergic Cell Death through Regulation of Microglial Metabolic Reprogramming.” Brain, Behavior & Immunity, vol. 60, Feb. 2017, pp. 206–19. EBSCOhost, https://doi.org/10.1016/j.bbi.2016.10.018.

APA

Gu, R., Zhang, F., Chen, G., Han, C., Liu, J., Ren, Z., Zhu, Y., Waddington, J. L., Zheng, L. T., & Zhen, X. (2017). Clk1 deficiency promotes neuroinflammation and subsequent dopaminergic cell death through regulation of microglial metabolic reprogramming. Brain, Behavior & Immunity, 60, 206–219. https://doi.org/10.1016/j.bbi.2016.10.018

Chicago

Gu, Ruinan, Fali Zhang, Gang Chen, Chaojun Han, Jay Liu, Zhaoxiang Ren, Yi Zhu, John L. Waddington, Long Tai Zheng, and Xuechu Zhen. 2017. “Clk1 Deficiency Promotes Neuroinflammation and Subsequent Dopaminergic Cell Death through Regulation of Microglial Metabolic Reprogramming.” Brain, Behavior & Immunity 60 (February): 206–19. doi:10.1016/j.bbi.2016.10.018.