Cite
Imatinib inhibits the regrowth of human colon cancer cells after treatment with 5-FU and cooperates with vitamin D analogue PRI-2191 in the downregulation of expression of stemness-related genes in 5-FU refractory cells.
MLA
Kotlarz, Agnieszka, et al. “Imatinib Inhibits the Regrowth of Human Colon Cancer Cells after Treatment with 5-FU and Cooperates with Vitamin D Analogue PRI-2191 in the Downregulation of Expression of Stemness-Related Genes in 5-FU Refractory Cells.” Journal of Steroid Biochemistry & Molecular Biology, vol. 189, May 2019, pp. 48–62. EBSCOhost, https://doi.org/10.1016/j.jsbmb.2019.02.003.
APA
Kotlarz, A., Przybyszewska, M., Swoboda, P., Neska, J., Miłoszewska, J., Grygorowicz, M. A., Kutner, A., & Markowicz, S. (2019). Imatinib inhibits the regrowth of human colon cancer cells after treatment with 5-FU and cooperates with vitamin D analogue PRI-2191 in the downregulation of expression of stemness-related genes in 5-FU refractory cells. Journal of Steroid Biochemistry & Molecular Biology, 189, 48–62. https://doi.org/10.1016/j.jsbmb.2019.02.003
Chicago
Kotlarz, Agnieszka, Małgorzata Przybyszewska, Paweł Swoboda, Jacek Neska, Joanna Miłoszewska, Monika Anna Grygorowicz, Andrzej Kutner, and Sergiusz Markowicz. 2019. “Imatinib Inhibits the Regrowth of Human Colon Cancer Cells after Treatment with 5-FU and Cooperates with Vitamin D Analogue PRI-2191 in the Downregulation of Expression of Stemness-Related Genes in 5-FU Refractory Cells.” Journal of Steroid Biochemistry & Molecular Biology 189 (May): 48–62. doi:10.1016/j.jsbmb.2019.02.003.