Cite
Endogenous AT1 receptor–protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension.
MLA
Ma, Huijie, et al. “Endogenous AT1 Receptor–protein Kinase C Activity in the Hypothalamus Augments Glutamatergic Input and Sympathetic Outflow in Hypertension.” Journal of Physiology, vol. 597, no. 16, Aug. 2019, pp. 4325–40. EBSCOhost, https://doi.org/10.1113/JP278427.
APA
Ma, H., Chen, S., Chen, H., & Pan, H. (2019). Endogenous AT1 receptor–protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension. Journal of Physiology, 597(16), 4325–4340. https://doi.org/10.1113/JP278427
Chicago
Ma, Huijie, Shao‐Rui Chen, Hong Chen, and Hui‐Lin Pan. 2019. “Endogenous AT1 Receptor–protein Kinase C Activity in the Hypothalamus Augments Glutamatergic Input and Sympathetic Outflow in Hypertension.” Journal of Physiology 597 (16): 4325–40. doi:10.1113/JP278427.