Cite
Calu-3 cells are largely resistant to entry driven by filovirus glycoproteins and the entry defect can be rescued by directed expression of DC-SIGN or cathepsin L.
MLA
González-Hernández, Mariana, et al. “Calu-3 cells Are Largely Resistant to Entry Driven by Filovirus Glycoproteins and the Entry Defect Can Be Rescued by Directed Expression of DC-SIGN or Cathepsin L.” Virology, vol. 532, June 2019, pp. 22–29. EBSCOhost, https://doi.org/10.1016/j.virol.2019.03.020.
APA
González-Hernández, M., Müller, A., Hoenen, T., Hoffmann, M., & Pöhlmann, S. (2019). Calu-3 cells are largely resistant to entry driven by filovirus glycoproteins and the entry defect can be rescued by directed expression of DC-SIGN or cathepsin L. Virology, 532, 22–29. https://doi.org/10.1016/j.virol.2019.03.020
Chicago
González-Hernández, Mariana, Andreas Müller, Thomas Hoenen, Markus Hoffmann, and Stefan Pöhlmann. 2019. “Calu-3 cells Are Largely Resistant to Entry Driven by Filovirus Glycoproteins and the Entry Defect Can Be Rescued by Directed Expression of DC-SIGN or Cathepsin L.” Virology 532 (June): 22–29. doi:10.1016/j.virol.2019.03.020.