miR-16-5p is upregulated by amyloid β deposition in Alzheimer's disease models and induces neuronal cell apoptosis through direct targeting and suppression of BCL-2.

MLA

Kim, Yeong-Jin, et al. “MiR-16-5p Is Upregulated by Amyloid β Deposition in Alzheimer’s Disease Models and Induces Neuronal Cell Apoptosis through Direct Targeting and Suppression of BCL-2.” Experimental Gerontology, vol. 136, July 2020, p. N.PAG. EBSCOhost, https://doi.org/10.1016/j.exger.2020.110954.

APA

Kim, Y.-J., Kim, S. H., Park, Y., Park, J., Lee, J. H., Kim, B. C., & Song, W. K. (2020). miR-16-5p is upregulated by amyloid β deposition in Alzheimer’s disease models and induces neuronal cell apoptosis through direct targeting and suppression of BCL-2. Experimental Gerontology, 136, N.PAG. https://doi.org/10.1016/j.exger.2020.110954

Chicago

Kim, Yeong-Jin, So Hee Kim, Yega Park, Jiyu Park, Jung Hoon Lee, Byeong C. Kim, and Woo Keun Song. 2020. “MiR-16-5p Is Upregulated by Amyloid β Deposition in Alzheimer’s Disease Models and Induces Neuronal Cell Apoptosis through Direct Targeting and Suppression of BCL-2.” Experimental Gerontology 136 (July): N.PAG. doi:10.1016/j.exger.2020.110954.