Cite
Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein.
MLA
Zhong, Jiahong, et al. “Roflupram Attenuates α-Synuclein-Induced Cytotoxicity and Promotes the Mitochondrial Translocation of Parkin in SH-SY5Y Cells Overexpressing A53T Mutant α-Synuclein.” Toxicology & Applied Pharmacology, vol. 436, Feb. 2022, p. N.PAG. EBSCOhost, https://doi.org/10.1016/j.taap.2021.115859.
APA
Zhong, J., Li, M., Xu, J., Dong, W., Qin, Y., Qiu, S., Li, X., & Wang, H. (2022). Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein. Toxicology & Applied Pharmacology, 436, N.PAG. https://doi.org/10.1016/j.taap.2021.115859
Chicago
Zhong, Jiahong, Mengfan Li, Jiangping Xu, Wenli Dong, Yunyun Qin, Shuqing Qiu, Xing Li, and Haitao Wang. 2022. “Roflupram Attenuates α-Synuclein-Induced Cytotoxicity and Promotes the Mitochondrial Translocation of Parkin in SH-SY5Y Cells Overexpressing A53T Mutant α-Synuclein.” Toxicology & Applied Pharmacology 436 (February): N.PAG. doi:10.1016/j.taap.2021.115859.