Cite
RGS3L allows for an M2 muscarinic receptor-mediated RhoA-dependent inotropy in cardiomyocytes.
MLA
Levay, Magdolna K., et al. “RGS3L Allows for an M2 Muscarinic Receptor-Mediated RhoA-Dependent Inotropy in Cardiomyocytes.” Basic Research in Cardiology, vol. 117, no. 1, Jan. 2022, pp. 1–21. EBSCOhost, https://doi.org/10.1007/s00395-022-00915-w.
APA
Levay, M. K., Krobert, K. A., Vogt, A., Ahmad, A., Jungmann, A., Neuber, C., Pasch, S., Hansen, A., Müller, O. J., Lutz, S., & Wieland, T. (2022). RGS3L allows for an M2 muscarinic receptor-mediated RhoA-dependent inotropy in cardiomyocytes. Basic Research in Cardiology, 117(1), 1–21. https://doi.org/10.1007/s00395-022-00915-w
Chicago
Levay, Magdolna K., Kurt A. Krobert, Andreas Vogt, Atif Ahmad, Andreas Jungmann, Christiane Neuber, Sebastian Pasch, et al. 2022. “RGS3L Allows for an M2 Muscarinic Receptor-Mediated RhoA-Dependent Inotropy in Cardiomyocytes.” Basic Research in Cardiology 117 (1): 1–21. doi:10.1007/s00395-022-00915-w.