Cite
The role of CD36-Fabp4-PPARγ in skeletal muscle involves insulin resistance in intrauterine growth retardation mice with catch-up growth.
MLA
Liu, Jing, et al. “The Role of CD36-Fabp4-PPARγ in Skeletal Muscle Involves Insulin Resistance in Intrauterine Growth Retardation Mice with Catch-up Growth.” BMC Endocrine Disorders, vol. 22, no. 1, Mar. 2022, pp. 1–12. EBSCOhost, https://doi.org/10.1186/s12902-021-00921-4.
APA
Liu, J., Zhao, H., Yang, L., Wang, X., Yang, L., Xing, Y., Lv, X., Ma, H., & Song, G. (2022). The role of CD36-Fabp4-PPARγ in skeletal muscle involves insulin resistance in intrauterine growth retardation mice with catch-up growth. BMC Endocrine Disorders, 22(1), 1–12. https://doi.org/10.1186/s12902-021-00921-4
Chicago
Liu, Jing, Hang Zhao, Linlin Yang, Xing Wang, Linquan Yang, Yuling Xing, Xiuqin Lv, Huijuan Ma, and Guangyao Song. 2022. “The Role of CD36-Fabp4-PPARγ in Skeletal Muscle Involves Insulin Resistance in Intrauterine Growth Retardation Mice with Catch-up Growth.” BMC Endocrine Disorders 22 (1): 1–12. doi:10.1186/s12902-021-00921-4.