Biochemical Fingerprints of Prion Infection: Accumulations of Aberrant Full-Length and N-Terminally Truncated PrP Species Are Common Features in Mouse Prion Disease.

MLA

Tao Pan, et al. “Biochemical Fingerprints of Prion Infection: Accumulations of Aberrant Full-Length and N-Terminally Truncated PrP Species Are Common Features in Mouse Prion Disease.” Journal of Virology, vol. 79, no. 2, Jan. 2005, pp. 934–43. EBSCOhost, https://doi.org/10.1128/JVI.79.2.934-943.2005.

APA

Tao Pan, Poki Wong, Binggong Chang, Chaoyang Li, Ruliang Li, Shin-Chung Kang, Wisniewski, T., & Man-Sun Sy. (2005). Biochemical Fingerprints of Prion Infection: Accumulations of Aberrant Full-Length and N-Terminally Truncated PrP Species Are Common Features in Mouse Prion Disease. Journal of Virology, 79(2), 934–943. https://doi.org/10.1128/JVI.79.2.934-943.2005

Chicago

Tao Pan, Poki Wong, Binggong Chang, Chaoyang Li, Ruliang Li, Shin-Chung Kang, Thomas Wisniewski, and Man-Sun Sy. 2005. “Biochemical Fingerprints of Prion Infection: Accumulations of Aberrant Full-Length and N-Terminally Truncated PrP Species Are Common Features in Mouse Prion Disease.” Journal of Virology 79 (2): 934–43. doi:10.1128/JVI.79.2.934-943.2005.