Phenotypic heterogeneity driven by plasticity of the intermediate EMT state governs disease progression and metastasis in breast cancer.

The article presents a study which explores the phenotypic heterogeneity driven by plasticity of the intermediate epithelial-to-mesenchymal transition (EMT) state governs disease progression and metastasis in breast cancer. It mentions that study reveals complex EMT spectrum and its regulatory networks, as well as the contributions of epithelial-mesenchymal plasticity (EMP) in tumor heterogeneity in breast cancer.