Cite
Fenretinide combines perturbation of signaling kinases, cell–extracellular matrix interactions and matrix metalloproteinase activation to inhibit invasion in oral squamous cell carcinoma cells.
MLA
Wang, Daren, et al. “Fenretinide Combines Perturbation of Signaling Kinases, Cell–extracellular Matrix Interactions and Matrix Metalloproteinase Activation to Inhibit Invasion in Oral Squamous Cell Carcinoma Cells.” Carcinogenesis, vol. 43, no. 9, Sept. 2022, pp. 851–64. EBSCOhost, https://doi.org/10.1093/carcin/bgac070.
APA
Wang, D., Pei, P., Shea, F. F., Bissonnette, C., Nieto, K., Din, C., Liu, Y., Schwendeman, S. P., Lin, Y. X., Spinney, R., & Mallery, S. R. (2022). Fenretinide combines perturbation of signaling kinases, cell–extracellular matrix interactions and matrix metalloproteinase activation to inhibit invasion in oral squamous cell carcinoma cells. Carcinogenesis, 43(9), 851–864. https://doi.org/10.1093/carcin/bgac070
Chicago
Wang, Daren, Ping Pei, Fortune F Shea, Caroline Bissonnette, Kari Nieto, Corrine Din, Yayuan Liu, et al. 2022. “Fenretinide Combines Perturbation of Signaling Kinases, Cell–extracellular Matrix Interactions and Matrix Metalloproteinase Activation to Inhibit Invasion in Oral Squamous Cell Carcinoma Cells.” Carcinogenesis 43 (9): 851–64. doi:10.1093/carcin/bgac070.