Cite
Songorine inhibits oxidative stress-related inflammation through PI3K/AKT/NRF2 signaling pathway to alleviate lipopolysaccharide-induced septic acute lung injury.
MLA
Fang, Jingjing, et al. “Songorine Inhibits Oxidative Stress-Related Inflammation through PI3K/AKT/NRF2 Signaling Pathway to Alleviate Lipopolysaccharide-Induced Septic Acute Lung Injury.” Immunopharmacology & Immunotoxicology, vol. 46, no. 2, Apr. 2024, pp. 152–60. EBSCOhost, https://doi.org/10.1080/08923973.2023.2281902.
APA
Fang, J., Huang, Q., Shi, C., Gai, L., Wang, X., & Yan, B. (2024). Songorine inhibits oxidative stress-related inflammation through PI3K/AKT/NRF2 signaling pathway to alleviate lipopolysaccharide-induced septic acute lung injury. Immunopharmacology & Immunotoxicology, 46(2), 152–160. https://doi.org/10.1080/08923973.2023.2281902
Chicago
Fang, Jingjing, Qin Huang, Chaolu Shi, Lei Gai, Xinnian Wang, and Biqing Yan. 2024. “Songorine Inhibits Oxidative Stress-Related Inflammation through PI3K/AKT/NRF2 Signaling Pathway to Alleviate Lipopolysaccharide-Induced Septic Acute Lung Injury.” Immunopharmacology & Immunotoxicology 46 (2): 152–60. doi:10.1080/08923973.2023.2281902.