RhoA/Rho kinase and nitric oxide modulate the agonist-induced pulmonary artery diameter response time.

Presents a study that postulated a role for RhoA/Rho kinase and nitric oxide (NO) in the modulation of pulmonary vascular reactivity. Role of myosin light chain phosphatase in the regulation of smooth muscle cell contraction; Vascular reactivity characteristics in endothelium-denuded arteries; Effects of endothelium removal and NO synthase inhibition on pulmonary vascular reactivity.