Cite
Both PI3K/Akt and ERK1/2 pathways participate in the protection by dexmedetomidine against transient focal cerebral ischemia/reperfusion injury in rats
MLA
Zhu, Yong-Man, et al. “Both PI3K/Akt and ERK1/2 Pathways Participate in the Protection by Dexmedetomidine against Transient Focal Cerebral Ischemia/Reperfusion Injury in Rats.” Brain Research, vol. 1494, Feb. 2013, pp. 1–8. EBSCOhost, https://doi.org/10.1016/j.brainres.2012.11.047.
APA
Zhu, Y.-M., Wang, C.-C., Chen, L., Qian, L.-B., Ma, L.-L., Yu, J., Zhu, M.-H., Wen, C.-Y., Yu, L.-N., & Yan, M. (2013). Both PI3K/Akt and ERK1/2 pathways participate in the protection by dexmedetomidine against transient focal cerebral ischemia/reperfusion injury in rats. Brain Research, 1494, 1–8. https://doi.org/10.1016/j.brainres.2012.11.047
Chicago
Zhu, Yong-Man, Can-Can Wang, Long Chen, Ling-Bo Qian, Lei-Lei Ma, Jing Yu, Man-Hua Zhu, Chuan-Yun Wen, Li-Na Yu, and Min Yan. 2013. “Both PI3K/Akt and ERK1/2 Pathways Participate in the Protection by Dexmedetomidine against Transient Focal Cerebral Ischemia/Reperfusion Injury in Rats.” Brain Research 1494 (February): 1–8. doi:10.1016/j.brainres.2012.11.047.