Cite
Overexpression of p62/SQSTM1 promotes the degradations of abnormally accumulated PrP mutants in cytoplasm and relieves the associated cytotoxicities via autophagy-lysosome-dependent way.
MLA
Xu, Yin, et al. “Overexpression of P62/SQSTM1 Promotes the Degradations of Abnormally Accumulated PrP Mutants in Cytoplasm and Relieves the Associated Cytotoxicities via Autophagy-Lysosome-Dependent Way.” Medical Microbiology & Immunology, vol. 203, no. 2, Apr. 2014, pp. 73–84. EBSCOhost, https://doi.org/10.1007/s00430-013-0316-z.
APA
Xu, Y., Zhang, J., Tian, C., Ren, K., Yan, Y.-E., Wang, K., Wang, H., Chen, C., Wang, J., Shi, Q., & Dong, X.-P. (2014). Overexpression of p62/SQSTM1 promotes the degradations of abnormally accumulated PrP mutants in cytoplasm and relieves the associated cytotoxicities via autophagy-lysosome-dependent way. Medical Microbiology & Immunology, 203(2), 73–84. https://doi.org/10.1007/s00430-013-0316-z
Chicago
Xu, Yin, Jin Zhang, Chan Tian, Ke Ren, Yu-E Yan, Ke Wang, Hui Wang, et al. 2014. “Overexpression of P62/SQSTM1 Promotes the Degradations of Abnormally Accumulated PrP Mutants in Cytoplasm and Relieves the Associated Cytotoxicities via Autophagy-Lysosome-Dependent Way.” Medical Microbiology & Immunology 203 (2): 73–84. doi:10.1007/s00430-013-0316-z.