Cite
Pressure overload induces IL-18 and IL-18R expression, but markedly suppresses IL-18BP expression in a rabbit model. IL-18 potentiates TNF-α-induced cardiomyocyte death.
MLA
Yoshida, Tadashi, et al. “Pressure Overload Induces IL-18 and IL-18R Expression, but Markedly Suppresses IL-18BP Expression in a Rabbit Model. IL-18 Potentiates TNF-α-Induced Cardiomyocyte Death.” Journal of Molecular & Cellular Cardiology, vol. 75, Oct. 2014, pp. 141–51. EBSCOhost, https://doi.org/10.1016/j.yjmcc.2014.07.007.
APA
Yoshida, T., Friehs, I., Mummidi, S., del Nido, P. J., Addulnour-Nakhoul, S., Delafontaine, P., Valente, A. J., & Chandrasekar, B. (2014). Pressure overload induces IL-18 and IL-18R expression, but markedly suppresses IL-18BP expression in a rabbit model. IL-18 potentiates TNF-α-induced cardiomyocyte death. Journal of Molecular & Cellular Cardiology, 75, 141–151. https://doi.org/10.1016/j.yjmcc.2014.07.007
Chicago
Yoshida, Tadashi, Ingeborg Friehs, Srinivas Mummidi, Pedro J. del Nido, Solange Addulnour-Nakhoul, Patrice Delafontaine, Anthony J. Valente, and Bysani Chandrasekar. 2014. “Pressure Overload Induces IL-18 and IL-18R Expression, but Markedly Suppresses IL-18BP Expression in a Rabbit Model. IL-18 Potentiates TNF-α-Induced Cardiomyocyte Death.” Journal of Molecular & Cellular Cardiology 75 (October): 141–51. doi:10.1016/j.yjmcc.2014.07.007.