A new receptor model-incremental lifetime cancer risk method to quantify the carcinogenic risks associated with sources of particle-bound polycyclic aromatic hydrocarbons from Chengdu in China.

PM 10 and PM 2.5 samples were simultaneously collected during a one-year monitoring period in Chengdu. The concentrations of 16 particle-bound polycyclic aromatic hydrocarbons (Σ 16 PAHs) were measured. Σ 16 PAHs concentrations varied from 16.85 to 160.24 ng m −3 and 14.93 to 111.04 ng m −3 for PM 10 and PM 2.5 , respectively. Three receptor models (principal component analysis (PCA), positive matrix factorization (PMF), and Multilinear Engine 2 (ME2)) were applied to investigate the sources and contributions of PAHs. The results obtained from the three receptor models were compared. Diesel emissions, gasoline emissions, and coal and wood combustion were the primary sources. Source apportionment results indicated that these models were able to track the ΣPAHs. For the first time, the cancer risks for each identified source were quantitatively calculated for ingestion and dermal contact routes by combining the incremental lifetime cancer risk (ILCR) values with the estimated source contributions. The results showed that gasoline emissions posed the highest cancer risk, even though it contributed less to Σ 16 PAHs. The results and method from this work can provide useful information for quantifying the toxicity of source categories and studying human health in the future. [ABSTRACT FROM AUTHOR]