Human placenta sphingomyelinase, an exogenous acidic pH-optimum sphingomyelinase, induces oxidative stress, glutathione depletion, and apoptosis in rat hepatocytes.

MLA

García-Ruiz, C., et al. “Human Placenta Sphingomyelinase, an Exogenous Acidic PH-Optimum Sphingomyelinase, Induces Oxidative Stress, Glutathione Depletion, and Apoptosis in Rat Hepatocytes.” Hepatology (Baltimore, Md.), vol. 32, no. 1, July 2000, pp. 56–65. EBSCOhost, https://doi.org/10.1053/jhep.2000.8267.

APA

García-Ruiz, C., Marí, M., Morales, A., Colell, A., Ardite, E., & Fernández-Checa, J. C. (2000). Human placenta sphingomyelinase, an exogenous acidic pH-optimum sphingomyelinase, induces oxidative stress, glutathione depletion, and apoptosis in rat hepatocytes. Hepatology (Baltimore, Md.), 32(1), 56–65. https://doi.org/10.1053/jhep.2000.8267

Chicago

García-Ruiz, C, M Marí, A Morales, A Colell, E Ardite, and J C Fernández-Checa. 2000. “Human Placenta Sphingomyelinase, an Exogenous Acidic PH-Optimum Sphingomyelinase, Induces Oxidative Stress, Glutathione Depletion, and Apoptosis in Rat Hepatocytes.” Hepatology (Baltimore, Md.) 32 (1): 56–65. doi:10.1053/jhep.2000.8267.