The cellular response to DNA damage induced by the enediynes C-1027 and neocarzinostatin includes hyperphosphorylation and increased nuclear retention of replication protein a (RPA) and trans inhibition of DNA replication.

MLA

McHugh, M. M., et al. “The Cellular Response to DNA Damage Induced by the Enediynes C-1027 and Neocarzinostatin Includes Hyperphosphorylation and Increased Nuclear Retention of Replication Protein a (RPA) and Trans Inhibition of DNA Replication.” Biochemistry, vol. 40, no. 15, Apr. 2001, pp. 4792–99. EBSCOhost, https://doi.org/10.1021/bi001668t.

APA

McHugh, M. M., Yin, X., Kuo, S. R., Liu, J. S., Melendy, T., & Beerman, T. A. (2001). The cellular response to DNA damage induced by the enediynes C-1027 and neocarzinostatin includes hyperphosphorylation and increased nuclear retention of replication protein a (RPA) and trans inhibition of DNA replication. Biochemistry, 40(15), 4792–4799. https://doi.org/10.1021/bi001668t

Chicago

McHugh, M M, X Yin, S R Kuo, J S Liu, T Melendy, and T A Beerman. 2001. “The Cellular Response to DNA Damage Induced by the Enediynes C-1027 and Neocarzinostatin Includes Hyperphosphorylation and Increased Nuclear Retention of Replication Protein a (RPA) and Trans Inhibition of DNA Replication.” Biochemistry 40 (15): 4792–99. doi:10.1021/bi001668t.