GRK6 deficiency is associated with enhanced CXCR4-mediated neutrophil chemotaxis in vitro and impaired responsiveness to G-CSF in vivo.

MLA

Vroon, Anne, et al. “GRK6 Deficiency Is Associated with Enhanced CXCR4-Mediated Neutrophil Chemotaxis in Vitro and Impaired Responsiveness to G-CSF in Vivo.” Journal of Leukocyte Biology, vol. 75, no. 4, Apr. 2004, pp. 698–704. EBSCOhost, https://doi.org/10.1189/jlb.0703320.

APA

Vroon, A., Heijnen, C. J., Raatgever, R., Touw, I. P., Ploemacher, R. E., Premont, R. T., & Kavelaars, A. (2004). GRK6 deficiency is associated with enhanced CXCR4-mediated neutrophil chemotaxis in vitro and impaired responsiveness to G-CSF in vivo. Journal of Leukocyte Biology, 75(4), 698–704. https://doi.org/10.1189/jlb.0703320

Chicago

Vroon, Anne, Cobi J Heijnen, Roel Raatgever, Ivo P Touw, Rob E Ploemacher, Richard T Premont, and Annemieke Kavelaars. 2004. “GRK6 Deficiency Is Associated with Enhanced CXCR4-Mediated Neutrophil Chemotaxis in Vitro and Impaired Responsiveness to G-CSF in Vivo.” Journal of Leukocyte Biology 75 (4): 698–704. doi:10.1189/jlb.0703320.