gamma-cleavage-independent functions of presenilin, nicastrin, and Aph-1 regulate cell-junction organization and prevent tau toxicity in vivo.

MLA

Doglio, Laura E., et al. “Gamma-Cleavage-Independent Functions of Presenilin, Nicastrin, and Aph-1 Regulate Cell-Junction Organization and Prevent Tau Toxicity in Vivo.” Neuron, vol. 50, no. 3, May 2006, pp. 359–75. EBSCOhost, https://doi.org/10.1016/j.neuron.2006.03.038.

APA

Doglio, L. E., Kanwar, R., Jackson, G. R., Perez, M., Avila, J., Dingwall, C., Dotti, C. G., Fortini, M. E., & Feiguin, F. (2006). gamma-cleavage-independent functions of presenilin, nicastrin, and Aph-1 regulate cell-junction organization and prevent tau toxicity in vivo. Neuron, 50(3), 359–375. https://doi.org/10.1016/j.neuron.2006.03.038

Chicago

Doglio, Laura E, Ritu Kanwar, George R Jackson, Mar Perez, Jesús Avila, Colin Dingwall, Carlos G Dotti, Mark E Fortini, and Fabián Feiguin. 2006. “Gamma-Cleavage-Independent Functions of Presenilin, Nicastrin, and Aph-1 Regulate Cell-Junction Organization and Prevent Tau Toxicity in Vivo.” Neuron 50 (3): 359–75. doi:10.1016/j.neuron.2006.03.038.