The -383A>C TNFRI polymorphism is associated with soluble levels and clinical activity in rheumatoid arthritis.

MLA

Valle, Y., et al. “The -383A>C TNFRI Polymorphism Is Associated with Soluble Levels and Clinical Activity in Rheumatoid Arthritis.” Rheumatology International, vol. 30, no. 5, Mar. 2010, pp. 655–59. EBSCOhost, https://doi.org/10.1007/s00296-009-1049-6.

APA

Valle, Y., Padilla-Gutiérrez, J. R., Torres-Carrillo, N. M., Ledezma-Lozano, I. Y., Corona-Sánchez, E. G., Vázquez-Del Mercado, M., Rangel-Villalobos, H., Gámez-Nava, J. I., González-López, L., & Muñoz-Valle, J. F. (2010). The -383A>C TNFRI polymorphism is associated with soluble levels and clinical activity in rheumatoid arthritis. Rheumatology International, 30(5), 655–659. https://doi.org/10.1007/s00296-009-1049-6

Chicago

Valle, Y, J R Padilla-Gutiérrez, N M Torres-Carrillo, I Y Ledezma-Lozano, E G Corona-Sánchez, M Vázquez-Del Mercado, H Rangel-Villalobos, J I Gámez-Nava, L González-López, and José Francisco Muñoz-Valle. 2010. “The -383A>C TNFRI Polymorphism Is Associated with Soluble Levels and Clinical Activity in Rheumatoid Arthritis.” Rheumatology International 30 (5): 655–59. doi:10.1007/s00296-009-1049-6.