The phosphatidylinositol 3-kinases (PI3K) inhibitor GS-1101 synergistically potentiates histone deacetylase inhibitor-induced proliferation inhibition and apoptosis through the inactivation of PI3K and extracellular signal-regulated kinase pathways.

MLA

Bodo, Juraj, et al. “The Phosphatidylinositol 3-Kinases (PI3K) Inhibitor GS-1101 Synergistically Potentiates Histone Deacetylase Inhibitor-Induced Proliferation Inhibition and Apoptosis through the Inactivation of PI3K and Extracellular Signal-Regulated Kinase Pathways.” British Journal of Haematology, vol. 163, no. 1, Oct. 2013, pp. 72–80. EBSCOhost, https://doi.org/10.1111/bjh.12498.

APA

Bodo, J., Zhao, X., Sharma, A., Hill, B. T., Portell, C. A., Lannutti, B. J., Almasan, A., & Hsi, E. D. (2013). The phosphatidylinositol 3-kinases (PI3K) inhibitor GS-1101 synergistically potentiates histone deacetylase inhibitor-induced proliferation inhibition and apoptosis through the inactivation of PI3K and extracellular signal-regulated kinase pathways. British Journal of Haematology, 163(1), 72–80. https://doi.org/10.1111/bjh.12498

Chicago

Bodo, Juraj, Xiaoxian Zhao, Arishya Sharma, Brian T Hill, Craig A Portell, Brian J Lannutti, Alexandru Almasan, and Eric D Hsi. 2013. “The Phosphatidylinositol 3-Kinases (PI3K) Inhibitor GS-1101 Synergistically Potentiates Histone Deacetylase Inhibitor-Induced Proliferation Inhibition and Apoptosis through the Inactivation of PI3K and Extracellular Signal-Regulated Kinase Pathways.” British Journal of Haematology 163 (1): 72–80. doi:10.1111/bjh.12498.