Loss of lysosomal membrane protein NCU-G1 in mice results in spontaneous liver fibrosis with accumulation of lipofuscin and iron in Kupffer cells.

MLA

Kong, Xiang Y., et al. “Loss of Lysosomal Membrane Protein NCU-G1 in Mice Results in Spontaneous Liver Fibrosis with Accumulation of Lipofuscin and Iron in Kupffer Cells.” Disease Models & Mechanisms, vol. 7, no. 3, Mar. 2014, pp. 351–62. EBSCOhost, https://doi.org/10.1242/dmm.014050.

APA

Kong, X. Y., Nesset, C. K., Damme, M., Løberg, E.-M., Lübke, T., Mæhlen, J., Andersson, K. B., Lorenzo, P. I., Roos, N., Thoresen, G. H., Rustan, A. C., Kase, E. T., & Eskild, W. (2014). Loss of lysosomal membrane protein NCU-G1 in mice results in spontaneous liver fibrosis with accumulation of lipofuscin and iron in Kupffer cells. Disease Models & Mechanisms, 7(3), 351–362. https://doi.org/10.1242/dmm.014050

Chicago

Kong, Xiang Y, Cecilie Kasi Nesset, Markus Damme, Else-Marit Løberg, Torben Lübke, Jan Mæhlen, Kristin B Andersson, et al. 2014. “Loss of Lysosomal Membrane Protein NCU-G1 in Mice Results in Spontaneous Liver Fibrosis with Accumulation of Lipofuscin and Iron in Kupffer Cells.” Disease Models & Mechanisms 7 (3): 351–62. doi:10.1242/dmm.014050.