Cytosolic H2O2 mediates hypertrophy, apoptosis, and decreased SERCA activity in mice with chronic hemodynamic overload.

MLA

Qin, Fuzhong, et al. “Cytosolic H2O2 Mediates Hypertrophy, Apoptosis, and Decreased SERCA Activity in Mice with Chronic Hemodynamic Overload.” American Journal of Physiology. Heart and Circulatory Physiology, vol. 306, no. 10, May 2014, pp. H1453–63. EBSCOhost, https://doi.org/10.1152/ajpheart.00084.2014.

APA

Qin, F., Siwik, D. A., Pimentel, D. R., Morgan, R. J., Biolo, A., Tu, V. H., Kang, Y. J., Cohen, R. A., & Colucci, W. S. (2014). Cytosolic H2O2 mediates hypertrophy, apoptosis, and decreased SERCA activity in mice with chronic hemodynamic overload. American Journal of Physiology. Heart and Circulatory Physiology, 306(10), H1453–H1463. https://doi.org/10.1152/ajpheart.00084.2014

Chicago

Qin, Fuzhong, Deborah A Siwik, David R Pimentel, Robert J Morgan, Andreia Biolo, Vivian H Tu, Y James Kang, Richard A Cohen, and Wilson S Colucci. 2014. “Cytosolic H2O2 Mediates Hypertrophy, Apoptosis, and Decreased SERCA Activity in Mice with Chronic Hemodynamic Overload.” American Journal of Physiology. Heart and Circulatory Physiology 306 (10): H1453–63. doi:10.1152/ajpheart.00084.2014.