The NOD2 p.Leu1007fsX1008 mutation (rs2066847) is a stronger predictor of the clinical course of Crohn's disease than the FOXO3A intron variant rs12212067.

MLA

Schnitzler, Fabian, et al. “The NOD2 p.Leu1007fsX1008 Mutation (Rs2066847) Is a Stronger Predictor of the Clinical Course of Crohn’s Disease than the FOXO3A Intron Variant Rs12212067.” PloS One, vol. 9, no. 11, Nov. 2014, p. e108503. EBSCOhost, https://doi.org/10.1371/journal.pone.0108503.

APA

Schnitzler, F., Friedrich, M., Wolf, C., Angelberger, M., Diegelmann, J., Olszak, T., Beigel, F., Tillack, C., Stallhofer, J., Göke, B., Glas, J., Lohse, P., & Brand, S. (2014). The NOD2 p.Leu1007fsX1008 mutation (rs2066847) is a stronger predictor of the clinical course of Crohn’s disease than the FOXO3A intron variant rs12212067. PloS One, 9(11), e108503. https://doi.org/10.1371/journal.pone.0108503

Chicago

Schnitzler, Fabian, Matthias Friedrich, Christiane Wolf, Marianne Angelberger, Julia Diegelmann, Torsten Olszak, Florian Beigel, et al. 2014. “The NOD2 p.Leu1007fsX1008 Mutation (Rs2066847) Is a Stronger Predictor of the Clinical Course of Crohn’s Disease than the FOXO3A Intron Variant Rs12212067.” PloS One 9 (11): e108503. doi:10.1371/journal.pone.0108503.