Eosinophil-specific deletion of IκBα in mice reveals a critical role of NF-κB-induced Bcl-xL for inhibition of apoptosis.

MLA

Schwartz, Christian, et al. “Eosinophil-Specific Deletion of IκBα in Mice Reveals a Critical Role of NF-ΚB-Induced Bcl-XL for Inhibition of Apoptosis.” Blood, vol. 125, no. 25, June 2015, pp. 3896–904. EBSCOhost, https://doi.org/10.1182/blood-2014-10-607788.

APA

Schwartz, C., Willebrand, R., Huber, S., Rupec, R. A., Wu, D., Locksley, R., & Voehringer, D. (2015). Eosinophil-specific deletion of IκBα in mice reveals a critical role of NF-κB-induced Bcl-xL for inhibition of apoptosis. Blood, 125(25), 3896–3904. https://doi.org/10.1182/blood-2014-10-607788

Chicago

Schwartz, Christian, Ralf Willebrand, Silke Huber, Rudolf A Rupec, Davina Wu, Richard Locksley, and David Voehringer. 2015. “Eosinophil-Specific Deletion of IκBα in Mice Reveals a Critical Role of NF-ΚB-Induced Bcl-XL for Inhibition of Apoptosis.” Blood 125 (25): 3896–3904. doi:10.1182/blood-2014-10-607788.