Cite
Nerve growth factor-induced Akt/mTOR activation protects the ischemic heart via restoring autophagic flux and attenuating ubiquitinated protein accumulation.
MLA
Wang, Zhou-Guang, et al. “Nerve Growth Factor-Induced Akt/MTOR Activation Protects the Ischemic Heart via Restoring Autophagic Flux and Attenuating Ubiquitinated Protein Accumulation.” Oncotarget, vol. 8, no. 3, Jan. 2017, pp. 5400–13. EBSCOhost, https://doi.org/10.18632/oncotarget.14284.
APA
Wang, Z.-G., Li, H., Huang, Y., Li, R., Wang, X.-F., Yu, L.-X., Guang, X.-Q., Li, L., Zhang, H.-Y., Zhao, Y.-Z., Zhang, C., Li, X.-K., Wu, R.-Z., Chu, M.-P., & Xiao, J. (2017). Nerve growth factor-induced Akt/mTOR activation protects the ischemic heart via restoring autophagic flux and attenuating ubiquitinated protein accumulation. Oncotarget, 8(3), 5400–5413. https://doi.org/10.18632/oncotarget.14284
Chicago
Wang, Zhou-Guang, Hao Li, Yan Huang, Rui Li, Xiao-Fan Wang, Li-Xia Yu, Xue-Qiang Guang, et al. 2017. “Nerve Growth Factor-Induced Akt/MTOR Activation Protects the Ischemic Heart via Restoring Autophagic Flux and Attenuating Ubiquitinated Protein Accumulation.” Oncotarget 8 (3): 5400–5413. doi:10.18632/oncotarget.14284.