Tissue-engineered smooth muscle cell and endothelial progenitor cell bi-level cell sheets prevent progression of cardiac dysfunction, microvascular dysfunction, and interstitial fibrosis in a rodent model of type 1 diabetes-induced cardiomyopathy.

MLA

Kawamura, Masashi, et al. “Tissue-Engineered Smooth Muscle Cell and Endothelial Progenitor Cell Bi-Level Cell Sheets Prevent Progression of Cardiac Dysfunction, Microvascular Dysfunction, and Interstitial Fibrosis in a Rodent Model of Type 1 Diabetes-Induced Cardiomyopathy.” Cardiovascular Diabetology, vol. 16, no. 1, Nov. 2017, p. 142. EBSCOhost, https://doi.org/10.1186/s12933-017-0625-4.

APA

Kawamura, M., Paulsen, M. J., Goldstone, A. B., Shudo, Y., Wang, H., Steele, A. N., Stapleton, L. M., Edwards, B. B., Eskandari, A., Truong, V. N., Jaatinen, K. J., Ingason, A. B., Miyagawa, S., Sawa, Y., & Woo, Y. J. (2017). Tissue-engineered smooth muscle cell and endothelial progenitor cell bi-level cell sheets prevent progression of cardiac dysfunction, microvascular dysfunction, and interstitial fibrosis in a rodent model of type 1 diabetes-induced cardiomyopathy. Cardiovascular Diabetology, 16(1), 142. https://doi.org/10.1186/s12933-017-0625-4

Chicago

Kawamura, Masashi, Michael J Paulsen, Andrew B Goldstone, Yasuhiro Shudo, Hanjay Wang, Amanda N Steele, Lyndsay M Stapleton, et al. 2017. “Tissue-Engineered Smooth Muscle Cell and Endothelial Progenitor Cell Bi-Level Cell Sheets Prevent Progression of Cardiac Dysfunction, Microvascular Dysfunction, and Interstitial Fibrosis in a Rodent Model of Type 1 Diabetes-Induced Cardiomyopathy.” Cardiovascular Diabetology 16 (1): 142. doi:10.1186/s12933-017-0625-4.