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Leigh syndrome-like MRI changes in a patient with biallelic HPDL variants treated with ketogenic diet.

Authors :
Numata-Uematsu Y
Uematsu M
Yamamoto T
Saitsu H
Katata Y
Oikawa Y
Saijyo N
Inui T
Murayama K
Ohtake A
Osaka H
Takanashi JI
Kure S
Inoue K
Source :
Molecular genetics and metabolism reports [Mol Genet Metab Rep] 2021 Sep 04; Vol. 29, pp. 100800. Date of Electronic Publication: 2021 Sep 04 (Print Publication: 2021).
Publication Year :
2021

Abstract

Biallelic 4-hydroxyphenylpyruvate dioxygenase-like protein ( HPDL ) variants were recently reported as a cause of progressive and incurable neurodegenerative diseases ranging from neonatal-onset leukoencephalopathy with severe neurodevelopmental delay to spastic paraplegia. Although the physiological function of HPDL remains unknown, its subcellular localization in the mitochondria has been reported. Here, we report a case of HPDL -related neurological disease that was clinically and neuroimaging compatible with Leigh syndrome, previously unreported, and was treated with a ketogenic diet.<br />Competing Interests: None.<br /> (© 2021 The Authors.)

Details

Language :
English
ISSN :
2214-4269
Volume :
29
Database :
MEDLINE
Journal :
Molecular genetics and metabolism reports
Publication Type :
Academic Journal
Accession number :
34522618
Full Text :
https://doi.org/10.1016/j.ymgmr.2021.100800