Canagliflozin, a SGLT-2 inhibitor, relieves ER stress, modulates autophagy and induces apoptosis in irradiated HepG2 cells: Signal transduction between PI3K/AKT/GSK-3β/mTOR and Wnt/β-catenin pathways; in vitro .

MLA

Abdel-Rafei, Mohamed Khairy, et al. “Canagliflozin, a SGLT-2 Inhibitor, Relieves ER Stress, Modulates Autophagy and Induces Apoptosis in Irradiated HepG2 Cells: Signal Transduction between PI3K/AKT/GSK-3β/MTOR and Wnt/β-Catenin Pathways; in Vitro .” Journal of Cancer Research and Therapeutics, vol. 17, no. 6, Oct. 2021, pp. 1404–18. EBSCOhost, https://doi.org/10.4103/jcrt.JCRT_963_19.

APA

Abdel-Rafei, M. K., Thabet, N. M., Rashed, L. A., & Moustafa, E. M. (2021). Canagliflozin, a SGLT-2 inhibitor, relieves ER stress, modulates autophagy and induces apoptosis in irradiated HepG2 cells: Signal transduction between PI3K/AKT/GSK-3β/mTOR and Wnt/β-catenin pathways; in vitro . Journal of Cancer Research and Therapeutics, 17(6), 1404–1418. https://doi.org/10.4103/jcrt.JCRT_963_19

Chicago

Abdel-Rafei, Mohamed Khairy, Noura Magdy Thabet, Laila Ahmed Rashed, and Enas Mahmoud Moustafa. 2021. “Canagliflozin, a SGLT-2 Inhibitor, Relieves ER Stress, Modulates Autophagy and Induces Apoptosis in Irradiated HepG2 Cells: Signal Transduction between PI3K/AKT/GSK-3β/MTOR and Wnt/β-Catenin Pathways; in Vitro .” Journal of Cancer Research and Therapeutics 17 (6): 1404–18. doi:10.4103/jcrt.JCRT_963_19.