Cite
Empagliflozin alleviates endoplasmic reticulum stress and augments autophagy in rotenone-induced Parkinson's disease in rats: Targeting the GRP78/PERK/eIF2α/CHOP pathway and miR-211-5p.
MLA
Motawi, Tarek K., et al. “Empagliflozin Alleviates Endoplasmic Reticulum Stress and Augments Autophagy in Rotenone-Induced Parkinson’s Disease in Rats: Targeting the GRP78/PERK/EIF2α/CHOP Pathway and MiR-211-5p.” Chemico-Biological Interactions, vol. 362, Aug. 2022, p. 110002. EBSCOhost, https://doi.org/10.1016/j.cbi.2022.110002.
APA
Motawi, T. K., Al-Kady, R. H., Abdelraouf, S. M., & Senousy, M. A. (2022). Empagliflozin alleviates endoplasmic reticulum stress and augments autophagy in rotenone-induced Parkinson’s disease in rats: Targeting the GRP78/PERK/eIF2α/CHOP pathway and miR-211-5p. Chemico-Biological Interactions, 362, 110002. https://doi.org/10.1016/j.cbi.2022.110002
Chicago
Motawi, Tarek K, Rawan H Al-Kady, Sahar M Abdelraouf, and Mahmoud A Senousy. 2022. “Empagliflozin Alleviates Endoplasmic Reticulum Stress and Augments Autophagy in Rotenone-Induced Parkinson’s Disease in Rats: Targeting the GRP78/PERK/EIF2α/CHOP Pathway and MiR-211-5p.” Chemico-Biological Interactions 362 (August): 110002. doi:10.1016/j.cbi.2022.110002.