1- O -Actylbritannilactone Ameliorates Alcohol-Induced Hepatotoxicity through Regulation of ROS/Akt/NF-κB-Mediated Apoptosis and Inflammation.

MLA

Xie, Li-Ya, et al. “1- O -Actylbritannilactone Ameliorates Alcohol-Induced Hepatotoxicity through Regulation of ROS/Akt/NF-ΚB-Mediated Apoptosis and Inflammation.” ACS Omega, vol. 7, no. 21, May 2022, pp. 18122–30. EBSCOhost, https://doi.org/10.1021/acsomega.2c01681.

APA

Xie, L.-Y., Yang, Z., Wang, Y., Hu, J.-N., Lu, Y.-W., Zhang, H., Jiang, S., & Li, W. (2022). 1- O -Actylbritannilactone Ameliorates Alcohol-Induced Hepatotoxicity through Regulation of ROS/Akt/NF-κB-Mediated Apoptosis and Inflammation. ACS Omega, 7(21), 18122–18130. https://doi.org/10.1021/acsomega.2c01681

Chicago

Xie, Li-Ya, Zhen Yang, Ying Wang, Jun-Nan Hu, Ya-Wei Lu, Hao Zhang, Shuang Jiang, and Wei Li. 2022. “1- O -Actylbritannilactone Ameliorates Alcohol-Induced Hepatotoxicity through Regulation of ROS/Akt/NF-ΚB-Mediated Apoptosis and Inflammation.” ACS Omega 7 (21): 18122–30. doi:10.1021/acsomega.2c01681.