A trans-kingdom T6SS effector induces the fragmentation of the mitochondrial network and activates innate immune receptor NLRX1 to promote infection.

MLA

Sá-Pessoa, Joana, et al. “A Trans-Kingdom T6SS Effector Induces the Fragmentation of the Mitochondrial Network and Activates Innate Immune Receptor NLRX1 to Promote Infection.” Nature Communications, vol. 14, no. 1, Feb. 2023, p. 871. EBSCOhost, https://doi.org/10.1038/s41467-023-36629-3.

APA

Sá-Pessoa, J., López-Montesino, S., Przybyszewska, K., Rodríguez-Escudero, I., Marshall, H., Ova, A., Schroeder, G. N., Barabas, P., Molina, M., Curtis, T., Cid, V. J., & Bengoechea, J. A. (2023). A trans-kingdom T6SS effector induces the fragmentation of the mitochondrial network and activates innate immune receptor NLRX1 to promote infection. Nature Communications, 14(1), 871. https://doi.org/10.1038/s41467-023-36629-3

Chicago

Sá-Pessoa, Joana, Sara López-Montesino, Kornelia Przybyszewska, Isabel Rodríguez-Escudero, Helina Marshall, Adelia Ova, Gunnar N Schroeder, et al. 2023. “A Trans-Kingdom T6SS Effector Induces the Fragmentation of the Mitochondrial Network and Activates Innate Immune Receptor NLRX1 to Promote Infection.” Nature Communications 14 (1): 871. doi:10.1038/s41467-023-36629-3.