Increased metabolism in combination with the novel cytochrome b target-site mutation L258F confers cross-resistance between the Q o inhibitors acequinocyl and bifenazate in Tetranychus urticae.

MLA

Lu, Xueping, et al. “Increased Metabolism in Combination with the Novel Cytochrome b Target-Site Mutation L258F Confers Cross-Resistance between the Q o Inhibitors Acequinocyl and Bifenazate in Tetranychus Urticae.” Pesticide Biochemistry and Physiology, vol. 192, May 2023, p. 105411. EBSCOhost, https://doi.org/10.1016/j.pestbp.2023.105411.

APA

Lu, X., Vandenhole, M., Tsakireli, D., Pergantis, S. A., Vontas, J., Jonckheere, W., & Van Leeuwen, T. (2023). Increased metabolism in combination with the novel cytochrome b target-site mutation L258F confers cross-resistance between the Q o inhibitors acequinocyl and bifenazate in Tetranychus urticae. Pesticide Biochemistry and Physiology, 192, 105411. https://doi.org/10.1016/j.pestbp.2023.105411

Chicago

Lu, Xueping, Marilou Vandenhole, Dimitra Tsakireli, Spiros A Pergantis, John Vontas, Wim Jonckheere, and Thomas Van Leeuwen. 2023. “Increased Metabolism in Combination with the Novel Cytochrome b Target-Site Mutation L258F Confers Cross-Resistance between the Q o Inhibitors Acequinocyl and Bifenazate in Tetranychus Urticae.” Pesticide Biochemistry and Physiology 192 (May): 105411. doi:10.1016/j.pestbp.2023.105411.