Complement C4-deficient mice have a high mortality rate during PTZ-induced epileptic seizures, which correlates with cognitive problems and the deficiency in the expression of Egr1 and other immediate early genes.

MLA

Veremeyko, Tatyana, et al. “Complement C4-Deficient Mice Have a High Mortality Rate during PTZ-Induced Epileptic Seizures, Which Correlates with Cognitive Problems and the Deficiency in the Expression of Egr1 and Other Immediate Early Genes.” Frontiers in Cellular Neuroscience, vol. 17, May 2023, p. 1170031. EBSCOhost, https://doi.org/10.3389/fncel.2023.1170031.

APA

Veremeyko, T., Jiang, R., He, M., & Ponomarev, E. D. (2023). Complement C4-deficient mice have a high mortality rate during PTZ-induced epileptic seizures, which correlates with cognitive problems and the deficiency in the expression of Egr1 and other immediate early genes. Frontiers in Cellular Neuroscience, 17, 1170031. https://doi.org/10.3389/fncel.2023.1170031

Chicago

Veremeyko, Tatyana, Rongcai Jiang, Mingliang He, and Eugene D Ponomarev. 2023. “Complement C4-Deficient Mice Have a High Mortality Rate during PTZ-Induced Epileptic Seizures, Which Correlates with Cognitive Problems and the Deficiency in the Expression of Egr1 and Other Immediate Early Genes.” Frontiers in Cellular Neuroscience 17 (May): 1170031. doi:10.3389/fncel.2023.1170031.