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Ocular neuromyotonia: an unusual case after radiotheraphy for nasopharyngeal carcinoma.
- Source :
-
Archivos de la Sociedad Espanola de Oftalmologia [Arch Soc Esp Oftalmol (Engl Ed)] 2024 May; Vol. 99 (5), pp. 209-212. Date of Electronic Publication: 2024 Feb 22. - Publication Year :
- 2024
-
Abstract
- Ocular neuromyotonia (ONM) is an infrequent disorder characterised by recurrent episodes of binocular diplopia caused by paroxysmal contraction of one or several extraocular muscles innervated by the same cranial nerve. It can be triggered spontaneously or caused by prolonged contraction of specific eye muscle(s) and is usually related to a local intracranial radiotherapy antecedent. We report the case of a 46-year-old woman who developed intermittent episodes of binocular diplopia eight years after radiotherapy for a nasopharyngeal carcinoma. After a complete neuro-ophthalmic assessment we diagnosed the case as an abducens nerve neuromyotonia. Although it is infrequent, radiotherapy to the nasopharynx is a possible cause of ONM, due to the proximity to the base of the skull and extraocular motor nerve pathways, especially that of the VI cranial nerve, as is the case presented in this article, about a patient whose history is a nasopharyngeal carcinoma treated with local radiotherapy.<br /> (Copyright © 2024 Sociedad Española de Oftalmología. Published by Elsevier España, S.L.U. All rights reserved.)
- Subjects :
- Humans
Female
Middle Aged
Carcinoma radiotherapy
Abducens Nerve Diseases etiology
Radiation Injuries etiology
Radiation Injuries complications
Radiotherapy adverse effects
Nasopharyngeal Neoplasms radiotherapy
Isaacs Syndrome etiology
Isaacs Syndrome diagnosis
Nasopharyngeal Carcinoma radiotherapy
Diplopia etiology
Subjects
Details
- Language :
- English
- ISSN :
- 2173-5794
- Volume :
- 99
- Issue :
- 5
- Database :
- MEDLINE
- Journal :
- Archivos de la Sociedad Espanola de Oftalmologia
- Publication Type :
- Academic Journal
- Accession number :
- 38401598
- Full Text :
- https://doi.org/10.1016/j.oftale.2024.01.017