Duodenal-jejunal bypass improves hypothalamic oxidative stress and inflammation in diabetic rats via glucagon-like peptide 1-mediated Nrf2/HO-1 signaling.

MLA

Wang, Huai-Jie, et al. “Duodenal-Jejunal Bypass Improves Hypothalamic Oxidative Stress and Inflammation in Diabetic Rats via Glucagon-like Peptide 1-Mediated Nrf2/HO-1 Signaling.” World Journal of Diabetes, vol. 15, no. 2, Feb. 2024, pp. 287–304. EBSCOhost, https://doi.org/10.4239/wjd.v15.i2.287.

APA

Wang, H.-J., Zhang, L.-B., Sun, S.-P., Yan, Q.-T., Gao, Z.-Q., Fu, F.-M., & Qu, M.-H. (2024). Duodenal-jejunal bypass improves hypothalamic oxidative stress and inflammation in diabetic rats via glucagon-like peptide 1-mediated Nrf2/HO-1 signaling. World Journal of Diabetes, 15(2), 287–304. https://doi.org/10.4239/wjd.v15.i2.287

Chicago

Wang, Huai-Jie, Li-Bin Zhang, Si-Peng Sun, Qing-Tao Yan, Zhi-Qin Gao, Fang-Ming Fu, and Mei-Hua Qu. 2024. “Duodenal-Jejunal Bypass Improves Hypothalamic Oxidative Stress and Inflammation in Diabetic Rats via Glucagon-like Peptide 1-Mediated Nrf2/HO-1 Signaling.” World Journal of Diabetes 15 (2): 287–304. doi:10.4239/wjd.v15.i2.287.